Inner ear hearing loss is called sensorineural hearing loss, or SNHL, which accounts for more than 90% of all hearing loss. Noise-induced hearing loss, NIHL, and age-related hearing loss, ARHL, account for a large majority of SNHL. Sadly, NIHL is happening to children and young adults more often recently. After 36 years of research. ACEMg from Soundbites can do something about the vast SNHL problem. You don’t need to believe us. The clinical data speaks for itself. The scientists at the U.S. Patent Office reviewed the data and granted our recent patent.

Recent auditory neuroscience goes beyond the phantom limb analogy.  Tinnitus and hyperacusis often reflect cochlear synaptopathy, or hidden hearing loss, HHL, which audiograms often miss. The root of HHL is stress at ribbon synapses linking inner hair cells to the auditory nerve, often from noise exposure, called noise-induced hidden hearing loss, NIHHL. Synaptic changes, essentially communication failures between sensory cells and the nerve fibers that send signals to the brain, frequently precede, and help explain, tinnitus and hyperacusis.

Age-related hearing loss is strongly linked to dementia risk. It increases social isolation and cognitive load—a processing bottleneck that slows the brain’s information pipeline and diverts resources from other tasks, causing mental fatigue. Mild loss may double risk; severe loss can raise it up to fivefold. Estimates of dementia cases attributable to hearing loss range from 9–32%, but even the lowest estimate is cause for concern. Hearing aids may slow decline, and preserving hearing in midlife is emerging as a leading preventive strategy.